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Table of contents
- Licence from the Régie du bâtiment - Services Québec Citoyens
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Stambolsky, P. Regulation of AIF expression by p Wang, D. Lebedeva, M. Loss of p53 causes mitochondrial DNA depletion and altered mitochondrial reactive oxygen species homeostasis. Saleem, A. Cell Physiol. Berkers, C. Metabolic regulation by p53 family members.
Licence from the Régie du bâtiment - Services Québec Citoyens
Ruiz-Lozano, P. Cell Growth Differ. Mathupala, S. Glucose catabolism in cancer cells. The type II hexokinase promoter contains functionally active response elements for the tumor suppressor p Jiang, P. Sahin, E. Telomere dysfunction induces metabolic and mitochondrial compromise. Goldstein, I. Cancer Metab.
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Wang, S. Cell Cycle 12 , — Zhang, P. Serine starvation induces stress and pdependent metabolic remodelling in cancer cells. This study describes a role for p53 in supporting metabolic adaptation to serine starvation by transiently activating p53, p21 and cell cycle arrest.
Potch doctor stripped of medical licence after series of botched surgeries
This enables depleted serine stores to be channelled to GSH synthesis, sustaining cellular redox balance and cell survival. The effectiveness of a serine-depleted diet in limiting cancer growth in vivo is also demonstrated. Reid, M. Locasale, J. Phosphoglycerate dehydrogenase diverts glycolytic flux and contributes to oncogenesis. Labuschagne, C. Serine, but not glycine, supports one-carbon metabolism and proliferation of cancer cells.
Cell Rep. Fan, J. The authors apply a novel and elegant technique, which uses liquid chromatography and mass spectrometry to track the source of NADPH production. Hanada, K. Serine palmitoyltransferase, a key enzyme of sphingolipid metabolism. Acta , 16—30 Chaneton, B.
police-risk-management.com/order/sms/kopef-programmi-per-spiare.php Serine is a natural ligand and allosteric activator of pyruvate kinase M2. Sanchez-Macedo, N. Depletion of the novel ptarget gene carnitine palmitoyltransferase 1C delays tumor growth in the neurofibromatosis type I tumor model. Assaily, W.
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ROS-mediated p53 induction of Lpin1 regulates fatty acid oxidation in response to nutritional stress. Cell 44 , — Currie, E. Cellular fatty acid metabolism and cancer. Choong, M. Specific activation of the p53 pathway by low dose actinomycin D: a new route to p53 based cyclotherapy. Cell Cycle 8 , — Choudhury, S. Dissecting the pathways that destabilize mutant p the proteasome or autophagy? Crighton, D. DRAM, a pinduced modulator of autophagy, is critical for apoptosis.
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Gao, W. Upregulation of human autophagy-initiation kinase ULK1 by tumor suppressor p53 contributes to DNA-damage-induced cell death. TRP53 activates a global autophagy program to promote tumor suppression. Autophagy 9 , — Lee, I.
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Atg7 modulates p53 activity to regulate cell cycle and survival during metabolic stress. Rao, S. A dual role for autophagy in a murine model of lung cancer. Rosenfeldt, M. This paper emphasizes the importance of determining the genetic make-up of a tumour before deciding on a treatment.
Using genetically engineered mice, the authors show that the p53 status of pancreatic tumours with activated oncogenic KRAS determines whether the inhibition of autophagy prevents or promotes tumour progression. Guo, J. Autophagy suppresses progression of K-ras-induced lung tumors to oncocytomas and maintains lipid homeostasis. Yang, A. Autophagy is critical for pancreatic tumor growth and progression in tumors with p53 alterations. Cancer Discov. Budanov, A. Feng, Z.
Tasdemir, E. Cell Cycle 7 , — Morselli, E. Cell Cycle 10 , — Livesey, K. Maillet, A.
Redox regulation of p53, redox effectors regulated by p a subtle balance. Redox Signal 16 , — Xie, J. TIGAR has a dual role in cancer cell survival through regulating apoptosis and autophagy.
Li, M. Cheung, E. Reilly, P.